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A new Method... San Diego, 2002

Prof.Bauer in Baar

Impresssum


16-9DVD
Prof. Bauer presents:
AUDIOVISUAL LECTURE
about Fibromyalgia on DVD.

Languages:
English, Deutsch, Français
Español, Italiano

NEW
More details? Click here ...
Example of a typical history
of generalized fibromyalgia
(all four quadrants affected).

Fibromyalgia (Fibromyalgia-Syndrom, FMS) - Healing is possible.
Now pain free, due to new methods of treatment: Surgical Quadrant-Pain-Intervention

Pathogenetic mechanisms in nerve pain
Source: Pathogenetische Mechanismen bei Nervenschmerzen, Beitrag M. Zimmermann „Physiologische Grundlagen des Schmerzes und der Schmerztherapie“, P. 3-13 in: Zenz-Jurna, Lehrbuch der Schmerztherapie, WVG, Stuttgart 1993
Nerve pain or neuropathic pain arises as a result of damage to peripheral nerves (Devor 1988), e.g. due to compression, transection or metabolic deficits (as in diabetes). Many somatic nerves pass through narrow anatomical sites where compression syndromes can occur under unfavourable conditions, e.g. carpal tunnel syndrome. The posterior spinal roots are also among the vulnerable sites; compression can occur here secondary to disc prolapse.
With more prolonged nerve compression or nerve transection, abnormal (ectopic) nerve fibre excitability develops (Howe et al. 1977, Culp and Ochoa 1982, Zimmermann et al. 1987, Bennet and Xie 1988, Jänig 1988). As a result, a variety of mechanical and chemical stimuli at the site of the nerve damage can stimulate afferent fibres. For instance, even adrenalin promotes excitation at the sprouts of regenerating nerves so the sympathetic nervous system can intensify pain associated with nerve injuries (see Chap. A2). The impulse patterns of ectopic excitation are irregular. They do not correspond to the impulses normally conducted in the nerve, e.g., those arising from a nociceptor. Patients have unnatural sensations and pain (Culp and Ochoa 1982, Zimmermann 1985).
After a nerve injury, the abnormalities do not remain limited to the site of the lesion but they spread slowly towards the periphery and spinal cord (Devor 1988). Non-electrical signal propagation also acts via axonal transport in the nerve fibre. This transport system works at speeds between 1 and 400 mm/day. Building and operating materials are transported from the cell body (protein synthesis) to the ends of the axons. Signal substances such as nerve growth factor (NFG), which are absorbed at the axon endings, reach the cell body by retrograde transport. There they can intervene to regulate biochemical functions. For instance, the synthesis of neuropeptides in the cell body of spinal ganglion neurons is altered after nerve transection (White and Zimmermann 1988). Prolonged expression of the nuclear protein c-Jun also occurs, a transcription factor that controls gene expression. In spinal neurons, transsynaptic induction of gene expression of neuropeptides and transcription factors has been observed after division of a peripheral nerve (see 1.5).
These findings show that local damage to the nervous system spreads slowly and extensively and so can trigger counter-regulatory and sometimes pathophysiological processes distant from the primary injury. This knowledge has not yet been exploited for pain therapy approaches to nerve injuries. *)

*) The widespread belief that fibromyalgia is difficult to understand, the problem needs to be explored is not true. Allegations that the FM is a rheumatic disease or even just imagined conflict, contradict clear the principle of modern medicine. This theoretical knowledge has been by Prof. Bauer realized, understood and he brought the basic parts in relation to each other. The scientific findings from the 80-ies of the last century were by him first implemented in 1990 in the practice. For over twenty years, thousands of patients have undergone the so-called "Quadrantenintervention" (surgical intervention in case of fibromyalgia) - with undreamt results.
Link to Download (PDF)


Do you question my method? That’s your good right!
But you should be consistent, and also question the methods offered by health insurance funds. Ask what studies have been performed proving that the proposed multimodal therapy, which is reimbursed by health insurance funds, makes fibromyalgia patients permanently pain free.
I know such patients don't exist. How could that be possible, when - according to current main stream medicine - fibromyalgia is incurable? If someone tells you about a patient that has become permanently pain free, please ask whether it is possible to get in touch with him/her in order to verify that statement.
All of my patients that are pain free are in contact with each other.
Fibromyalgia is a perpetual motion machine, in desire by the pharmaceutical industry, supported by doctors and condoned by health insurance funds.
 


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